Hyperaldosteronism

Common Name(s)

Hyperaldosteronism

Description for this condition is not yet available.
 

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Advocacy and Support Organizations

 

Condition Specific Organizations

Following organizations serve the condition "Hyperaldosteronism" for support, advocacy or research.

There are currently no organizations listed in Disease InfoSearch that support this condition. Create a listing.

 

 

General Support Organizations

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Scientific Literature

Articles from the PubMed Database

Research articles describe the outcome of a single study. They are the published results of original research.
The terms "Hyperaldosteronism" returned 128 free, full-text research articles on human participants. First 3 results:

Histopathological Classification of Cross-Sectional Image-Negative Hyperaldosteronism.
 

Author(s): Yuto Yamazaki, Yasuhiro Nakamura, Kei Omata, Kazue Ise, Yuta Tezuka, Yoshikiyo Ono, Ryo Morimoto, Yukinaga Nozawa, Celso E Gomez-Sanchez, Scott A Tomlins, William E Rainey, Sadayoshi Ito, Fumitoshi Satoh, Hironobu Sasano

Journal: J. Clin. Endocrinol. Metab.. 2017 04;102(4):1182-1192.

 

Approximately half of patients with primary aldosteronism (PA) have clinically evident disease according to clinical (hypertension) and/or laboratory (aldosterone and renin levels) findings but do not have nodules detectable in routine cross-sectional imaging. However, the detailed ...

Last Updated: 31 Dec 1969

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A Novel Phenotype of Familial Hyperaldosteronism Type III: Concurrence of Aldosteronism and Cushing's Syndrome.
 

Author(s): Anli Tong, Guanghua Liu, Fen Wang, Jun Jiang, Zhaoli Yan, Dianxi Zhang, Yinsheng Zhang, Jun Cai

Journal: J. Clin. Endocrinol. Metab.. 2016 11;101(11):4290-4297.

 

To date, all the familial hyperaldosteronism type III (FH-III) patients reported presenting with typical primary aldosteronism (PA), without showing other adrenal hormone abnormalities.

Last Updated: 31 Dec 1969

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Disordered zonal and cellular CYP11B2 enzyme expression in familial hyperaldosteronism type 3.
 

Author(s): Celso E Gomez-Sanchez, Xin Qi, Elise P Gomez-Sanchez, Hironobu Sasano, Martin O Bohlen, Max Wisgerhof

Journal: Mol. Cell. Endocrinol.. 2017 Jan;439():74-80.

 

Three forms of familial primary aldosteronism have been recognized. Familial Hyperaldosteronism type 1 (FH1) or dexamethasone suppressible hyperaldosteronism, FH2, the most common form of as yet unknown cause(s), and FH3. FH3 is due to activating mutations of the potassium channel ...

Last Updated: 31 Dec 1969

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Reviews from the PubMed Database

Review articles summarize what is currently known about a disease. They discuss research previously published by others.
The terms "Hyperaldosteronism" returned 7 free, full-text review articles on human participants. First 3 results:

Arterial Hypertension, Atrial Fibrillation, and Hyperaldosteronism: The Triple Trouble.
 

Author(s): Teresa M Seccia, Brasilina Caroccia, Gail K Adler, Giuseppe Maiolino, Maurizio Cesari, Gian Paolo Rossi

Journal: Hypertension. 2017 04;69(4):545-550.

 

Last Updated: 31 Dec 1969

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Genetics of primary hyperaldosteronism.
 

Author(s): Ravi Kumar Dutta, Peter Söderkvist, Oliver Gimm

Journal: Endocr. Relat. Cancer. 2016 10;23(10):R437-54.

 

Hypertension is a common medical condition and affects approximately 20% of the population in developed countries. Primary aldosteronism is the most common form of secondary hypertension and affects 8-13% of patients with hypertension. The two most common causes of primary aldosteronism ...

Last Updated: 31 Dec 1969

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Mutations in KCNJ5 gene cause hyperaldosteronism.
 

Author(s): Maria-Christina Zennaro, Xavier Jeunemaitre

Journal: Circ. Res.. 2011 Jun;108(12):1417-8.

 

Last Updated: 31 Dec 1969

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Clinical Trial Information This information is provided by ClinicalTrials.gov

Primary Aldosteronism in Western Norway
 

Status: Recruiting

Condition Summary: Hyperaldosteronism

 

Last Updated: 11 Aug 2017

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Chongqing Primary Aldosteronism Study
 

Status: Recruiting

Condition Summary: Primary Aldosteronism

 

Last Updated: 19 Jul 2017

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Macrolides for KCNJ5 - Mutated Aldosterone-Producing Adenoma (MAPA)
 

Status: Not yet recruiting

Condition Summary: Hyperaldosteronism

 

Last Updated: 29 Jan 2018

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